III Increased left ventricular torsion in hypertrophic cardiomyopathy mutation
carriers with normal wall thickness Wessel P. Brouwer*, Iris K. Rüssel*, Tjeerd Germans, Paul Knaapen, J. Tim Marcus, Jolanda van der Velden, Marco J.W. Götte, Albert C. van Rossum
Abstract Background Increased left ventricular (LV) torsion has been observed in patients with manifest
familial hypertrophic cardiomyopathy (HCM), and is thought to be caused by
subendocardial dysfunction. We hypothesize that increased LV torsion is already present in healthy mutation carriers with normal wall thickness. Methods